- Here is some of the remarkable information from Dr. Wolff
Geisler's website regarding both the origin of BSE diseases and also the
origin of AIDS.
-
- Patricia Doyle
-
- For More Information:
- http://members.aol.com/GeislerW/index.htm
- http://members.aol.com/GeislerW/bse/bse.htm
-
-
- Mass-diseases of Bovine Spongiform Encephalopathy
- (Mad Cow Disease) are caused by artificial spreading
- of retroviral particles bred in Campylobacter microbes
-
- 1 Bovine Spongiform Encephalopathy (BSE) was not introduced
by food prepared out of sheep
- Since several decades meat and bonemeal from sheep and
cattle have been fed to cattle and sheep without causing BSE.
-
- Since its official recognition in 1986 in Britain BSE
has in not one single study been induced in cattle by feeding commercially
sold feedstuff made from sheep or any other commercially available material.
[1] "...no evidence of an association with any single compounder of
proprietary feedstuffs... all had received proprietary feed-stuffs."
[2]
-
- None of the companies selling the material has been made
responsible for causing the mass-slaughter of 180,000 cows.
-
- There are no comparable numbers of diseases in the north
of France and in Denmark, even though the same feedstuff from United Kingdom
is sold there.
-
- Hundreds of cases of BSE occure in cattle in Switzerland
(220 cases [3]), Portugal (940 inborne cattle plus 300, which had been
imported from Britain [4]), France and Ireland without connection to feedstuff
from United Kingdom.
-
- The appearance of BSE diseases was an simultaneous emergence
in a large number of flocks, in defined regions of the United Kingdom.
[5] Absolutely and percentagewise rarely affected were cattle herds of
farmers in the counties Buckinghamshire and East-Sussex, Durham, Yorkshire
South, Derbyshire, Merseyside and Essex, in Northern Ireland, Scotland,
and in Isle of Man. [6]
- The diseases were allegedly provoked by "commercially
induced changes in rendering of abattoir, butchers and knackers waste,
resulting collectively from a fall in the value of tallow, a rise in the
cost of energy and a need to replace old plant with safer systems...".
[7]
-
- No single details of the pretended changes are given.
The accused changes were not introduced in 1981 but in the late sixties.
-
- According to scientific articles the agent causing BSE
can only be inactivated by heating up to 180 or even 270º Celsius
[8] and hardly by chemical means. In Scotland few cattle were affected
by BSE, because allegedly the old plants were not replaced.
-
- In reality the agent is inactivated by the old and the
new methods. The World Health Organization (WHO) f.e. considers gelatine,
produced from infectious bones, to be safe for medical capsules. The product
only allows a processing temperature of 140º Celsius. The theory of
S.B. Prusiner of the undetected prions transmitted in food and in genetic
material is stated without any proof and contradicting basic laws of nature
f.e. of the physico-chemical binding capacities in proteins. It can not
be followed.
-
- The British government could have reintroduced the former
- the Scotish - extraction methods in the whole United Kingdom.
-
- Allegedly the mass diseases started in 1981, because
cattle were exposed to meal made from Scrapie-infected brain from sheeps.
To fulfill that condition, there must have been a manyfold stronger increase
in Scrapie of sheep before the BSE wave. There are no statistics which
would underline that. The available numbers of Scrapie and the respective
numbers of BSE are: [9]
-
- Scrapie BSE
- 1980 104 -
- 1981 112 -
- 1982 139 -
- 1983 155 -
- 1984 169 -
- 1985 149 15
- 1986 156 63
- 1987 179 662
- 1988 215 2184
- 1989 246 7137
- 1990 348 14181
- 1991 989 25032
- 1992 666 36681
- 1993 93 34370
- 1994 73 23944
- 1995 75 14282
-
- And if there was an increase of Spongiform Encephalopathy
among sheep, it is unexplained, what has caused that increase?
-
- But there was also, at the same time, when the cattle
were infected, a mass infection of sheep. (See chapter 3.4)
-
- In 1996, eight years after the ban of feeding the accused
material to ruminants, 300 cases of BSE/week are occuring in the United
Kingdom. The majority of them are descendants born after 1992 to cows who
themselves were born after the implementation of the feedban.
-
- Contrary to the impression which the British government
tries to create, today the diseases are transmitted to a large extent from
cows to calves. [10] In Portugal in 1994 the great-granddaughter of a cow
imported from Great Britain was affected by BSE. [11] But even the British
government concedes, that at a non significant part BSE is transmitted
by this route.
-
- By this claim about the significant part of transmission
in food produced before the feedban in 1988 the British government expresses,
that thousands of British farmers bought at the time of the feedban in
advance food for many years and intentionally infect their cattle; and
that the cattle eat that horribly smelling material today.
-
- On 1st August 1996 the British Ministry of Agriculture,
Fisheries and Food quoted the findings of the Spongiform Encephalopathy
Advisory Committee (SEAC), SEAC statement on maternal transmission of BSE
... Of the 273 animals born to dams with BSE, 42 have developed histologically
confirmed BSE. [12] This is a 15.4% transmission-rate from cows with BSE
to calves. The SEAC quoted by the British government converts this fact:
...the average transmission from cow to calf over the 60 months duration
of infection in an animal prior to developing clinical disease will be
1%. [13]
-
- 3 Mass diseases affecting brain and immunosystem like
BSE in other animals and in other areas
-
- 3.1 Cows in Great Britain
-
- Other mass-diseases of cattle in United Kingdom must
be seen connected with occurence of BSE. [1] Of 24,000 cows slaughtered
in 1994 in United Kingdom because of symptoms suspicious of BSE 25% did
not have pathological findings of the brain indicative of BSE. These 6,000
were affected by neurological symptoms and immunodeficiency. Especially
in March/April diseases reminding of BSE but without visible lesions in
the brain are observed. [2] They correspond to f.e. Downer Cows in the
USA (see chapter 3.2). Since about 1977 there was an increase of lameness
of dairy cows connected with lactation, especially occuring in March and
April, in the same regions of the British island [3] where later increased
BSE incidences appeared.
-
- 3.2 Cows in USA, Canada
-
- In the USA annually about 100,000 cattle are slaughtered
because of a brain damaging [4] neurological disease named Downer Cow Syndrome.
This disease affects milk fever cows. It occurs in heavy milking cows...
related to the calving period and if after futile treatment with calcium
hypocalcaemia is excluded as causing disorder the hind legs... extended...
posteriorally... hyperaesthesia... and.... non-alert... of this Downer
Cows [5] should be compared with the BSE-symptoms. Contrary to claims of
the USA-government at a considerable part these syndromes are the result
of causes comparable to those of BSE (see chapter 6). In 1985 mink in Wisconsin
were affected by an outbreak of Spongiform Encephalopathy caused by consumption
of Downer Cows. [6]
-
- Two bull calves, which were infected intracerebrally
with brain of the diseased mink developed Spongiform Encephalopathy. [7]
-
- The clinical signs of Downer Cow Syndrome were presented
by cattle inoculated intracerebrally in 1979 with Scrapie-agents in Mission,
Texas. When slaughtered 43, 44, or 73 days later their brain did not (yet?;
a.) show the histological findings of Spongiform Encephalopathy. But inoculated
into mice it produced central nervous system signs in them. And tissue
from the cattle were found to be positive for prion protein [8], indicative
of Spongiform Encephalopathy.
-
- In 1993 the Canadian government ordered to slaughter
270 cows on one ranch in Alberta, and 30 others on nine farms in Ontario.
The pretended reason was, that BSE was diagnosed in one cow in Alberta.
This cow was allegedly of British origin. [9]
-
- 3.3 Cows in Germany
-
- In 1988 9,000 calves had immediately to be slaughtered
in Germany under emergency circumstances. The government claimed, that
they had been treated with too many hormones. [10] The calves had even
to be incinerated. Hormones are easily removed by the body in a few weeks.
So the hormones were the pretext for other findings.
-
- 3.4 Sheep in Scotland, Germany, Australia, and USA
-
- The slaughtering of sheep in Northengland and Scotland
1986 till 1988 was already mentioned. From summer 1986 till July 1988 herds
of sheep in Scotland and in the north of Britain had to be slaughtered
on command of the British government; and they became forbidden for human
consumption in the United Kingdom: Their stomach was said, to contain grass
contaminated by radiation from Czernobyl. [11] This given reason is invalid,
because it is possible to measure radiation and to select animals accordingly.
By radiation contaminated grass in the stomach does not make a sheep unfit
for human consumption. Possibly the sheep were affected by the same causes
of Spongiform Encephalopathy like the cattle at the same time.
-
- 1,200 sheep were slaughtered in July 1990 in Germany
because of Scrapie [12], 240 in the year 1991 [13].
-
- Sheeps in Australia are dying allegedly because of eating
a new plant. [14]
-
- Between 1984 and 1988 in the USA the number of Scrapie
outbreaks in herds of sheep has increased by ten times compared to before
1979. [15]
-
- 3.5 Elk and deer in zoos in USA, ready for export
-
- Since 1967 in Fort Collins, Colorado, in the Wild Animal
Disease Center of the USA-army cofinanced by the British Burroughs Wellcome
Company and than in Sybille Wildlife Research Unit, Wyoming deers died
and since 1979, especially since 1984, elks died there as well as in connected
wildlife research facilities in Kremmling, Colorado, in Meeker, Colorado,
in a further zoo in Colorado, in Wheatland, Wyoming, in a further zoo in
Wyoming and in a zoo in Canada. They died by a new Spongiform Encephalopathy
named Chronic Wasting Disease of Deer and Elk (CWD).
-
- 90% of the deers in Fort Collins died or were killed,
mainly 3-4 years old. Most affected were female elk. The origin of this
disease has not been published. But in experimental feeding trials they
were fed with cow's milk starting at 1-5 days of age. The similarity of
the disease with one caused by a neurotropic retrovirus ... in wild mice
is mentioned in the reporting articles.
-
- A possible effect of the diseases has been described:
CWD should be of concern in interstate and international trade in captive
cervids. [16]
-
- 3.6 Elk in Sweden, deers in Austria
-
- From 1985 till 1995 mostly female elk, in the southwest
of Sweden bordering Lake Vaenern, died in masses following neurological
and immunodeficiency symptoms caused by retroviruses. In 1994 this disease
was observed also among roedeers and deers. [17]
-
- Since December 1986 masses of young deers died in Austria
showing neurological symptoms and necrotic lesions of the brain. [18]
-
- 3.7 Seals, wales, dolphins, seacrows
-
- In 1988, 18,000 seals died on European coasts showing
neurological symptoms, wales died around New Foundland, Canada and around
Sovjet-Union-Russia, dolphins around the North-American coasts. [19] Seacrows
died in masses in Florida, in 1990 and 1996. The Armed Forces Institute
of Pathology of the USA military forces took care of the dead animals which
had shown neuromuscular signs before death. The incidence in 1990 was explained
by cold water stress and crashes with motorboats. [20] The incidences in
1996 are allegedly mysterious.
-
- 3.8 Tropical animals in zoos in England, Germany, and
USA affected by Spongiform Encephalopathy
-
- Since 1983 in zoos in England 6% of all Nyala-antelopes,
11% of Eland-antelopes in one zoo together with a mouflon and an Ugandan
Ankole-cow and two domestic cats, 35% of Greater Kudu-antelopes (62% of
those born since 1987), an Arabian Oryx cow and her calf (see following),
a Scimitary-horned Oryx, a puma and four cheetahs and in a German zoo three
ostriches died because of Spongiform Encephalopathy. They were percentagewise
much stronger affected than the British cows [21] (less than 2% in 1996).
-
- 3.9 Lions, tigers, leopards in zoos in USA and Western-Europe
affected by encephalopathies and retroviruses.
-
- 50 lions, three tigers, and one puma died between 1969
and end of 1972 because of an unknown infectious encephalomeningitis in
a safari park in Germany. They had been fed with meat of horses and of
cows. [22] In zoos in the USA (see chapter 7) died, because of unexplained
neurological symptoms in 1982 two tigers, and in 1988 two leopards in Iowa,
in 1991 two leopards and a jaguar in Illinois.[24] In 1989 ten lions in
a Safari Park in Germany died of similar neurological symptoms [23]. In
1992, in a zoo in San Fernando, California 35 big cats showed strange neurological
symptoms. Of them died seven African lions, four Asian tigers, four African
leopards, one Chinese leopard, and one jaguar. Other indigenous North American
cats were unaffected. In 1992 five tigers and one lion in a zoo in Acton,
California showed the same neurological symptoms: two of the tigers died.
Even though the symptoms of all the cats in Europe and USA were unexplained,
exclusively in only one lion of all those diseased cats an electro encephalogram
and an analysis of the cerebrospinal fluid was done. [24] The electro encephalograms
of Spongiform Encephalopathies show typical changes.
-
- 57% of tested African lions in European zoos were infected
by the retrovirus Feline Immunodeficiency Virus (FIV), but 0% of Asiatic
lions in India. [25] (Spongiform Encephalopathy is caused by particles
of retroviruses, see chapter 6.) But 73% of Asiatic lions in the Lincoln
Park Zoo in Chicago had Feline Immunodeficiency Virus-antibodies. [26]
... FIV infection ... It may well be that the high prevalence ... is caused
by a factor associated with the zoo., suspected the author of the article.
-
- In 41 of 109 tested big cats in the zoos in Europe antibodies
against the Virus of Infectious Anaemia of Horses (EIAV) were present.
(EIAV is the original virus of HIV [27]). Antibodies to EIAV were found
in most ... of the FIV positive animals ... indicates that there may exist
an additional feline lentivirus. [28] In one region in Florida 30% of panthers
captured between 1986 - 1988 had FIV antibodies. [29]
-
- 3.10 Export from English zoos to African countries and
the Persian Gulf area
-
- Obviously English zoos functioned as breeding places
for animals containing agents of Spongiform Encephalopathy to transmit
them into parts of Africa and into Oman and Saudi Arabia. London zoo officials
have written to other British zoos suggesting that `careful consideration'
should be given before animals at risk are exported to foreign zoos or
for reintroduction programmes. London zoo's circular says that any ungulate
fed properietary meal between 1980 and 1989 may be affected. [30]
-
- "There is growing interest in the reintroduction
of captive-bred animals to the wild for conservation programmes and these
reintroductions provide a route for the accidental transmission of infectious
diseases into free-living populations..." [31].
-
- In April 1989 an Arabian oryx cow died in Londons Regent's
Park because of BSE. Three month later, in July 1989, her nine month old
calf, was shipped to Saudi Arabia. He died there on Aug 18, 1989. [32]
In Phoenix, Arizona, USA 216 Arabian oryx are awaiting to be shipped to
Saudi Arabia.
-
- In Oman, the import of the BSE agents from Great Britain
was kept hidden for four years. In Jan./Feb. 1989 two cows there showed
clinical symptoms indicative of BSE. They had been exported from one affected
herd in Great Britain in 1985. Their BSE-illnesses were the first cases
to be diagnosed outside Great Britain. [33] From January 1988 to March
1989 fully vaccinated children in Oman were attacked by an outbreak of
paralytic poliomyelitis [34], indicative of immunosuppression. Soldiers
from the USA, being active there at that time, became victims of Gulf War
Syndrome, a disease indicative of retrovirus-particles connected with BSE.
(See chapter 5)
-
- In Serengeti Park in Tanzania in 1994 during 14 days
over an 1,000 km distance 1,000 out of 3,000 lions died showing neurological
symptoms [35] reminding of Spongiform Encephalopathy. The lions are affected
by the retrovirus Feline Immunodeficiency virus [36] (see chapter 3.9).
95% of the young cheetahs there did not survive in 1994. [37] 80% of the
lions in Kruger National Park, South Africa, bordering to Mozambique, are
infected by Feline Immunodeficiency Virus. [38]
-
- Jackals and foxes since 1978, wild dogs since 1991, uncounted
hyaenas, foxes and leopards died in East Africa, showing neurological symptoms
like the lions.
-
- In 1994 50% of the buffalos and 25% of gnus in Serengeti
died, the surviving are in a bad health condition.
-
- Imported and domestic cows are dying in Uganda [39].
-
- Dromedars in the north of Somalia are showing Bovine
Spongiform Encephalopathy-symptoms. [40]
-
- Measles-viruses are in most of the cases named in articles
in scientific papers to be responsible for those neurological and immunosuppressing
diseases of seals, wales, dolphins, of elk, of buffalos, gnus, of lions,
leopards, cheetahs, jaguars, jackals, hyaenas, and foxes. But they were
never proven to be the causes for these mass-diseases. And they are unlikely
to have mutated at so many places in the USA, England, Germany, Tanzania,
Kenya and on coasts in North-America and Europe to cross the species barrier
to such an extent and at so many places. For millions of years they had
not affected f.e. cats. So we have to count for an immunosppressive agent
enabling the infection of measles viruses.
-
- 5 BSE-agents cause also Chronic Fatigue Syndrome or Gulf
War Syndrome
-
- In the United Kingdom in same regions, where BSE is prevailing
[1], in 1988 about 100,000, in 1996 150,000, persons were reported to be
affected by a mysterious, new disease affecting brain, spinal cord and
immunofunctions called mostly Chronic Fatigue Syndrome. In the USA in the
HIV-regions California-Nevada, Boston, New York, Houston, and Florida since
1984 five million persons are reported to be affected. Clusters were reported
in 1985 for Tuscon, Arizona [2], Lake Tahoe, Nevada (more than 400 patients
1984 till 1988 [3]), Lyndonville, N.Y. (rural, transmission in raw milk
since 1979 [4]), for Charlotte, North Carolina, for soldiers from USA stationed
in the Persian Gulf Area. It was reported among persons in Natal, South
Africa in 1987. (Kwazulu in Natal experiences today the worst HIV/AIDS
explosion in South Africa.) It is also called Chronic Fatigue Immune Dysfunction
Syndrome (CFIDS), or Post Viral Fatigue Syndrome (PVFS), or Myalgic Encephalomyelitis
(ME), or Gulf War Syndrome. Fatigue, depression, poor concentration, loss
of memory, impaired balance, are the reported neurological symptoms, and
fever, sore throat, cough, night sweats, painful lymph-nodes, weakness,
pain of muscles, rash caused by viruses and fungi are indicating an impaired
function of T-lymphocytes. The disease starts with a flu-like illness with
respiratory or gastro-intestinal symptoms.
-
- ....of affected persons are women about 20 to 45 years
old. White persons are eight times more frequent affected than non-white.
[5] (See chapter 6.7)
-
- --------------------------------------------------------------------------------
-
-
- 6 Bovine Spongiform Encephalopathy, Downer Cow Syndrome,
Creutzfeldt-Jakob Disease, Scrapie, Chronic Fatigue Syndrome, and AIDS
are caused by exogenous and endogenous parts of retroviruses
-
- 6.1 Retroviruses cause Spongiform Encephalopathies
- By direct effect of retroviral particles on neurons Spongiform
Encephalopathy is evoked. [1]
-
- Spongiform Encephalopathy is caused in mice by a retrovirus.
[2]
-
- 6.2 Structures, indicative for retroviral infection,
existing in all cases of Spongiform Encephalopathies
- Tubulofilamentous (virus-like) particles are seen under
electron microscope in all cases of Spongiform Encephalopathies in the
brains of all affected animals and human beings.
-
- In human beings they had been observed beside Creutzfeldt-Jakob
Disease-Kuru only extremely seldom; namely in the brain of some persons
with Systemic Lupus Erythematosus, in Encephalomyelitis disseminata, in
HTLV-1-infection, in cases of leukaemia. This are all diseases at least
suspicious for retroviral involvement. Since 1983 it became known, that
lymphocytes, makrophages and endothel cells of every person with AIDS contained
this tubulofilamentous particles [3] which have to be considered subviral
structures... defective viruses [4].
-
- 6.3 Creutzfeldt-Jakob Disease connected with retroviral
particles
- The agent of Creutzfeldt-Jakob Disease has a virus like
size and density. [5]
-
- The agent of Creutzfeldt-Jakob Disease is a viral oncogenic
particle. [6]
-
- The similarities of Creutzfeldt-Jakob Disease and infections
of central nervous system by retroviruses like HIV were outlined by E.
and L. Manuelidis. They point out that CJD-like agents might contain truncated
or mutated retroviral sequences. Alternatively, the CJD agent might require
`helper' functions of an endogenous retrovirus [7]. As outlined for HIV
among haemophilia-patients the necessary cofactor for such endogenous retroviral
particles could be Parvovirus B19. [8] The same might apply to Spongiform
Encephalopathies. Aleutian Mink Disease underlines that.
-
- 6.4 Scrapie connected with retrovirus HIV
- Parts of HTLV-3 (HIV) and Scrapie are identical. [9]
Other researchers were diminishing these findings. The relationship between
PrP27-30 is at best distant... [10], but they admit the similarities.
-
- 6.5 Chronic Fatigue Syndrome connected with retroviral
particles and effects
- Sections of the genetic material of the human retrovirus
HTLV-2, a close relative of the AIDS-virus, were present in Chronic Fatigue
Syndrome patients and non-sexual contacts, but not in controls. [11] Some
authors tried to create a diverging impression. For example Gow et al.
selected as controls persons with diseases, which are itself suspicious
of retroviral effects like Systemic Lupus Erythematosus, Rheumatoid Arthritis,
Epilepsy, Migraine. Finding retroviral particles in the Chronic Fatigue
Syndrome patients and in these controls, they claim these to be endogenous
retroviral sequences, without any reason. [12]
-
- The existence of a virus containing sequences of both
herpes virus and retrovirus in persons with CFS was proven with PCR (Polymerase
Chain Reaction; a.) technique in 1991. [13] (Emphasis by a.)
-
- In Japan, where HTLV-1 and HTLV-2 are prevailing endemically,
30 Chronic Fatigue Syndrome-patients showed a weak reaction comparable
to the healthy controls. But in Western immunoblotting 16 patients with
Chronic Fatigue Syndrome showed clearly positive lanes in rgp21, gp21,
env gp46, and gag p26, indicative of HTLV-2 seropositivity. They were different
from the healthy controls which showed nothing. [14] The article tries
to create a different impression.
-
- In the muscle of patients with Postviral Fatigue Syndrome
seven times more often than in controls persistency of enteroviruses proves
the involvment of a retrovirus or endogenous retroviral sequences [15]
evoking immunosuppression.
-
- In Jamaica the existence of HTLV-1, another close relative
of the AIDS-virus, was proven in persons with pains of muscles, impaired
balance, weakness [16], resembling to Myalgic Encephalomyelitis.
-
- D. Diack reported to have found by electron microscopic
studies particles similar to the ultrastructure of Visna Virus (retrovirus)
in lymphocytes of patients with Chronic Fatigue Syndrome. [17]
-
- Research suggests that ME (Myalgic Encephalomyelitis;
a.) is the result of a persistent infection, probably caused by a virus.
[18]
-
- The abnormal results of the MRI (magnetic resonance imaging;
a.) scans observed among these persons... similar... in a population positive
for human immunodeficiency virus [19].
-
- 6.6 Several types of retroviruses in respective mass-diseaes
- Particles of different types of retroviruses are occuring
in AIDS (HIVEIAV and HTLV-1 and HTLV-2), in Chronic Fatigue Syndrome (HIVEIAV
and HTLV-1 and HTLV-2) in Creutzfeldt-Jakob Disease/Kuru (HIVEIAV and Scrapie-agent),
and in Downer Cow Syndrome (BLV and Scrapie-agent and EIAV).
-
- 6.7 Identity of retroviral amino-acid sequences and amino-acid
sequences of messenger substances of animals and humans
- Important biochemical effects of HIV-retrovirus are outlined
in a book [20]. They are described here again, to show the parallels between
biochemically induced hypothalamic effects of HIV and of the agents of
Chronic Fatigue Syndrome, of Creutzfeldt-Jakob Disease, of Downer Cow Syndrome,
and of Scrapie:
-
- HIV attacks the body's forces of defence in two places:
-
- One amino acid sequence of the core-structure of HIV
coincides in 44-50% with the amino acid sequence in one part of the hormone
alpha1-thymosine-alpha1, which is produced by the human thymus gland. This
hormone is required for the formation of the helper cells. When infected
with HIV, the body incorrectly assumes, that it has sufficient alpha1-thymosine-alpha1:
new thymosine is only produced to a lesser extent with the result that
fewer helper cells are produced [21]. Over and above that, antibodies formed
by the body to fight the HIV then go into action, destroying both the HIV,
which has penetrated from the outside, and also the body's own alpha1-thymosine-alpha1
[22].
- On the other hand, an amino acid sequence of the HIV-envelope
(gp 120) equals the amino acid sequence of the Vasoactive intestinal peptide
(VIP) formed in intestinal cells [23]. This hormone is a messenger substance
which regulates the release of important hormones, including Corticotropin
Releasing-Hormone (CRH), which in relation to AIDS, is of decisive importance:
CRH encourages production of cortisol.
- ... Cortisol triggers off a vicious circle
-
- Cortisol is a hormone produced by the human body which
counteracts inflammations and allergies. Among others, it restricts helper
cells activity [24] and transfers them from the active blood-circulation
to the dormant area of the bone marrow [25]. Where the hormone-balance
is stable, a daily rhythm regulates the cortisol level in the blood, which
in turn determines the daily rhythm of the helper cells in the blood [26].
The cortisol level is highest at around six a.m.; by lunch time it has
dropped sharply and only starts to rise again slowly from midnight onwards.
The cortisol level in the blood of HIV-infected patients is higher than
that of non-infected persons. 16 tests involving HIV patients yielded this
result. Four study-groups showed normal amounts of cortisol, whilst only
one out of the 21 groups of scientists involved in this topic, up to 1990,
found smaller amounts of cortisol in the blood of HIV-infected patients
[27]. All examinations based on the 24-hour rhythm produced higher values
[28]. There are fewer helper cells as a result of the raised cortisol level
in the blood of patients affected by AIDS or who are in a possibly preliminary
stage of the disease. Of decisive significance is the changing of the rhythm
of cortisol-production [29]. The cortisol level of AIDS patients is constantly
slightly raised, even during the night. (Such a change in cortisol-rhythm
was already described in 1977 by physicians working in the Mulago hospital
in Kampala, Uganda. What the twelve patients examined were actually suffering
from, was not divulged [30].) A sustained increase of cortisol, to a small
degree, produces a particularly marked reduction in the quantity of T cells
in the blood [31].
-
- The Human Immunodeficiency Virus manages to upset the
central regulation. The organism is taken in by the structure of HIV: On
one hand it figures to face neuroleukin VIP [32], which is produced by
the helper cells [33], and on the other hand it figures to face its own
hormone alpha1-thymosine-alpha1, while in reality it is facing HIV-core-protein.
Deception of the immunosystem thus sets in motion a chain-reaction: the
Corticotropin-Releasing Hormone (CRH) yielded and scattered in error leads
to increased release of the Adrenocorticotropic Hormone (ACTH). Because
of the ACTH, the adrenal glands increase the production of cortisol [34].
The cortisol then brings the organism to a state of partial immunodeficiency:
agents of the AIDS diseases can now strike because the immunoreaction to
unknown agents is impaired. (Against well-known agents several protecting
mechanisms, which can not be influenced by cortisol, were developed.) Neuroleukin
- or rather HIV-gp120 in the case of HIV patients - leads also to a release
in growth hormones, watery diarrhoea, outbreaks of sweating, fever, and
increased immunoglobulin synthesis [35].
-
- An effect of feedback in healthy subjects is, that cortisol
impedes the production of the CRH signalling substance which has set this
mechanism in motion. However, with AIDS, the feedback effect is cancelled
out once again because of production of ACTH at other places: leukocytes
and lymphocytes being attacked by the virus release ACTH, which stimulates
cortisol-production [36]. And HIV-infected monocytes stimulate even by
themselves the adrenal glands to produce cortisol [37]. HIV increases the
amount of cortisol. And cortisol accelerates as a growth substance for
HIV [38].
-
- We see as HIV-effect an increase of neuroleukin - VIP
- type sequences of eight, respectively five amino-acids, which evoke Growth
hormone.
-
- Growth hormone identical sequences can stimulate protein
synthesis and are multiplied under the conditions of growth. (Growth hormone
deficient persons in Central Africa, so called Pygmies are unexpected rarely
infected by HIV [39].)
-
- Comparable effects are produced by agents of Spongiform
Encephalopathy-diseases.
-
- The agent of Creutzfeldt-Jakob Disease evokes secretion
of Growth Factor out of infected cells. [40]
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- Retroviral amino-acid sequences can stimulate lactation,
and they can be multiplied under the conditions of lactation.
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- Affected cows (Downer Cows;a.) had significantly increased
plasma levels of cortisol, fatty acids,... [41] high cortisol levels in
blood [42] significantly lower erythrocyte... concentrations. [43]
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- Patients with Chronic Fatigue Syndrome have enhanced
release of ACTH and enhanced sensitivity to exogenous ACTH. [44] But values
for Cortisol are diminished.
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- DHEA is decreased in Chronic Fatigue Syndrome like in
AIDS patients. [45]
- EIAV (HIV) is enhanced by deficiency of calcium. [46]
Downer Cow Syndrome is like-wise enhanced by deficiency of calcium. This
is connected with the functioning of the Reverse Transcriptase.
- People of Jewish (and Italian) origin are preferred by
attacks of agents causing AIDS diseases like Kaposi Sarcoma, Pneumocystis
Carinii, as well as by agents causing Creutzfeldt-Jakob Disease. HIV prefers
white persons before black persons [47], the same do the agents of Chronic
Fatigue Syndrome.
- 15% of cases of Scrapie and of Creutzfeldt-Jakob Disease
are caused by inherited agents. 12.8% of children of mothers with HIV-infection
are born with HIV. 15.4% of calves of BSE-cows are born with BSE-agents
according to the British SEAC. [48] This figures signal similarity too.
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- Patricia A. Doyle, PhD
- Please visit my "Emerging Diseases" message
board at: http://www.clickitnews.com/ubbthreads/postlist.php?Cat=&Board=emergingdiseases
- Zhan le Devlesa tai sastimasa
- Go with God and in Good Health
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