- According to a time-line of physicians and therapists
reports, posted on The Empire Journal, Terri's injury occurred on 2/25/90:
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- 2/25/1990 Terri's Injury
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- 2/25/1990 X-Ray Report - Dr. Hameroff Images taken of
cervical spine, no acute bony pathology - straightening of normal cervical
lordosis
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- 2/25/1990 Neuro Consult - Dr. DeSousa Deeply comatose.
No evidence of acute process in CT scan. Evidence of myoclonic seizures.
Rule out acute myocardiac infarction, seizures as cause. Neck is somewhat
stiff as is all of the muscles of the body. No jugular venous distension.
WBC 26,300, drug screen negative
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- 2/25/1990 CT Scan Report - Dr. Hameroff Normal
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- 2/26/1990 - EEG Report - Dr. DeSousa Abnormal EEG indicative
of generalized suppression and slowing
-
- 2/27/1990 - DeSousa Report Brainstem Auditory Evoked
Response shows no significant disruption of the brain stem acoustic pathway
- study within normal limits
-
- 2/27/1990 - CT Scan Report - Dr. Greenberg Normal CT
Scan
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- 2/28/1990 - EEG Report - Dr. DeSousa Generalized diffuse
slowing. Some fast frequency rhythms which were not obvious during previous
recording. May have been slight improvement over last EEG.
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- 3/19/1990 - EEG Report - Dr. DeSousa Markedly abnormal
EEG, no significant improvement from previous records
-
- 3/30/1990 - CT Scan Report - Dr. Abramson CT Scan shows
noncommunicating hydrocephalus, changes occurred since 2/27 exam
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- If you look carefully at this time line you will see
that Terri had "NORMAL" CT's of the brain on 2/25 and 2/27.
-
- STOP THE PRESSES
-
- There is categorically and absolutely NO WAY Terri could
have suffered MASSIVE ANOXIC INJURY TO THE BRAIN ON 2/25 AND HAVE NORMAL
CT SCAN ON 2/27.
-
- NO WAY.
-
- Brain edema begins to occur about 20 minutes after infarction
and by 24 hours her brain (if she HAD suffered a massive anoxic event)
would have been MASSIVELY SWOLLEN -- something that could NEVER be missed
and NEVER called normal.
-
- NEVER.
-
- If Terri's brain CT was NORMAL 2 days after she entered
the hospital than there is NO POSSIBLE WAY she suffered a massive infarction
or global ischemia on 2/25.
-
- THIS IS ALL WRONG
-
- Now look at 3/30. Suddenly she develops NONCOMMUNICATING
HYDROCEPHALUS.
-
- WHAT?
-
- Did anyone ask HOW? How did she develop noncommunicating
hydrocephalus suddenly on 3/30/90 with 2 normal CT scans on 2/235 and 2/27??
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- CONCLUSIONS?
-
- 1. IF this is an accurate report (normal CT brain on
2/27 -- injury on 2/25) then TERRI DID NOT suffer an event of massive ischemia
on 2/25. THERE IS NO RADIOLOGIST OR NEUROLOGIST OR NEUROSURGEON IN THE
WORLD THAT WOULD DISPUTE THIS. It is impossible. The CT on 2/27 would have
been GROSSLY abnormal.
-
- 2. IF TERRI DID NOT SUFFER ANOXIC DAMAGE ON 2/25 THEN
THE REASON FOR HER BRAIN ATROPHY WAS CAUSED BY SOMETHING THAT OCCURRED
after 2/25 namely in the hospital during February or March of 1990.
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- 3. How does one develop NONCOMMUNICATING HYDROCEPHALUS
in ONE MONTH? By a blood clot obstructing the CSF outflow from the brain
at the Foramen of Magendie.
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- 4. How does one get #3.
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- BY BEING HIT ON THE HEAD AND SUFFERING INTRACRANIAL HEMORRHAGE.
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- So Terri WAS HIT ON THE HEAD OR DROPPED ON HER HEAD DURING
LATER FEBRUARY OR EARLY MARCH WHILE IN THAT HOSPITAL.
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- http://codeblueblog.blogs.com/
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