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USDA Fights Court Decision
Approving BSE Tests

From Terry S. Singeltary Sr.
flounder9@verizon.net
5-30-7

To: agsec@usda.gov
Cc: john.clifford@aphis.usda.gov; usaha@usaha.org; jmeng@cpfbeef.com;LAVET22@aol.com Phyllis.Fong@usda.gov
Sent: Tuesday, May 29, 2007 2:07 PM
Subject: USDA VS CREEKSTONE BSE/BASE/TSE TESTING Civil Action No. 06-0544
(JR)
 
 
May 27, 2007
 
Honorable Michael Johanns
Secretary of Agriculture
U.S. Department of Agriculture
Room 200 Jamie Whitten Federal Building
Washington, D.C. 20250
 
CC
 
Honorable Judge James Robertson
U.S. District Court
333 Constitution Ave. North West
Washington, D. C. 20001
 
Subject: Request to let the Creekstone vs. USDA court decision stand.
 
Ref: Letter from United States Animal Health Association, dated May 22,
2007
 
Dear Mr. Secretary et al :
 
I am requesting that you allow the court decision in the Creekstone vs. USDA to stand so that Creekstone may begin testing the beef they process for BSE and or BASE and or any other TSE phenotype there of. WE must let them test since the USDA et al refuse to do so properly. This is not to say that there should be no strict TSE testing protocols. IF testing is to take place privately, there must be strict TSE testing protocol to assure the most up to date, sensitive, and validated tests are used, and used properly. These tests must be announced to the public in a timely manner at every step of the way, validated and confirmed by the federal government, Weybridge, and an independent third party consumer organization and there TSE expert of choice, in my opinion.
 
My mother died from a exceedingly rare strain of sporadic CJD i.e. the Heidenhain Variant of CJD. My neighbors mother also lost his mother to a form of sporadic CJD exactly one year previously from the day my mother died. BOTH cases were confirmed by autopsy. There is new data out about the BASE atypical BSE, which pathologically is more related to a phenotype of sporadic CJD, than the nvCJD in humans from the UK. To continue to ignore these scientific findings with the old UKBSEnvCJD only theory is not justified by science anymore. It is not logical.
 
The logic behind the reasons not to let test for TSE in the USA because of The Virus Serum Toxin Act of 1913 and or because of the recent letter from the USAHA (see letter below) bring forth, are totally bogus. NO one could screw the testing up any worse than the USDA has done in the past. The OIG and the GAO has shown this time and time again. The 2004 Enhanced BSE surveillance program where some 275,000+ cattle were tested for BSE was proven to be terribly flawed from the beginning. This documented time and time again. Even Paul Brown, known and respected TSE scientist, former TSE expert for the CDC said he had ''absolutely no confidence in USDA tests before one year ago'', and this was on March 15, 2006 ;
 
"The fact the Texas cow showed up fairly clearly implied the existence of other undetected cases," Dr. Paul Brown, former medical director of the National Institutes of Health's Laboratory for Central Nervous System Studies and an expert on mad cow-like diseases, told United Press International. "The question was, 'How many?' and we still can't answer that."
 
Brown, who is preparing a scientific paper based on the latest two mad cow cases to estimate the maximum number of infected cows that occurred in the United States, said he has "absolutely no confidence in USDA tests before one year ago" because of the agency's reluctance to retest the Texas cow that initially tested positive.
 
USDA officials finally retested the cow and confirmed it was infected seven months later, but only at the insistence of the agency's inspector general.
 
"Everything they did on the Texas cow makes everything USDA did before 2005 suspect," Brown said. ...snip...end
 
http://www.upi.com/ConsumerHealthDaily/view.php?StoryID=20060315-055557-1284r
 
CDC - Bovine Spongiform Encephalopathy and Variant Creutzfeldt ... Dr. Paul Brown is Senior Research Scientist in the Laboratory of Central Nervous System ... Address for correspondence: Paul Brown, Building 36, Room 4A-05, ...
 
http://www.cdc.gov/ncidod/eid/vol7no1/brown.htm
 
PAUL BROWN COMMENT TO ME ON THIS ISSUE
 
Tuesday, September 12, 2006 11:10 AM
 
"Actually, Terry, I have been critical of the USDA handling of the mad cow issue for some years, and with Linda Detwiler and others sent lengthy detailed critiques and recommendations to both the USDA and the Canadian Food Agency."
 
OR, what the Honorable Phyllis Fong of the OIG found ;
 
 
Audit Report
Animal and Plant Health Inspection Service
Bovine Spongiform Encephalopathy (BSE) Surveillance Program ­ Phase II
and
Food Safety and Inspection Service
 
Controls Over BSE Sampling, Specified Risk Materials, and Advanced Meat
Recovery Products - Phase III
 
Report No. 50601-10-KC January 2006
 
Finding 2 Inherent Challenges in Identifying and Testing High-Risk Cattle
Still Remain
 
http://www.usda.gov/oig/webdocs/50601-10-KC.pdf
 
Mr. Johanns,
 
The August 4, 1997 FDA BSE ruminant to ruminant feed ban was nothing more than ink on paper. In 2007 alone, 10 MILLION plus pounds of banned blood laced MBM has already gone out into commerce for the feeding of banned product to cattle. yes, were still feeding cows banned BSE/BASE product in 2007, almost 10 years after the voluntary ban was put in place. guess what, it aint working.
 
YOU and this Administration have failed terribly in protecting not only the consumer, but your precious commodity that you speak so highly of i.e. the beef industry. In your continued efforts to cover up the real mad cow problem in the USA, you have in fact only amplified it and continued it's spread, and in doing so, you have needlessly exposed millions to the TSE agent, from many different proven routes and sources. The only saving grace you have is the incubation period has been on your side. It will catch up. When it does, when the people finally figure all this out, when some of the millions you have needlessly exposed to this agent become clinical in the future, rest assured I will stand in line to see that you and your administration are convicted for murder.
 
What you and this administration have done over the past 8 years is criminal, in my opinion. I have watched not only you, but the Bush administration thumb there nose to science for almost 8 years, all to protect the beef industry. The science was there, but you chose to ignore it, and even manipulated science with the bogus BSE MRR policy, all the while your were implementing that, you were covering up another mad cow in Texas. But thanks to the Honorable Phyllis Fong of the OIG, and an act of Congress, that mad cow was finally proven positive, unlike the other stumbling and staggering mad cow that was rendered without any test at all in Texas, but by then you had succeeded in the BSE MRR policy, the legal trading of all strains of TSE globally. You and this administration have done the same thing the UK did when they poisoned the globe with there exporting of BSE, except you made it legal now with the BSE MRR policy, and now we are dealing with BASE, a strain that is more virulent to humans. what happens when it mutates again?
 
When cwd deer and elk and there different phenotypes have all been rendered into feed, along with scrapie infected sheep in the USA, and a few TME to top that off, it will be a most interesting recipe will it not, and an interesting case study for humans for decades to come. sadly though, with the recent pet food scandal, and the deaths there of, we have learned a few things. one, that the elderly are expendable, but cats, dogs, and adolescents are not. and that the problem of our feeding of food producing animals has been tainted for decades. and with the melamine scandal, as with the mad cow feed scandal, it's the same old song and dance by you and the Bush administration, everything is o.k., will not hurt you, cover-up and protect the industry at all cost, and this will be another part of your sad legacy in History Sir.
 
To not allow BSE/TSE testing in the USA, testing that will find, only proves our point, you have and will continue to cover up the real mad cow problem in the USA. and the world knows this. ...
 
Terry S. Singeltary Sr.
P.O. Box 42
Bacliff, Texas USA 77519
 
UNITED STATES ANIMAL HEALTH ASSOCIATION
8100 Three Chopt Road, Suite 203
P. O. BOX K227
RICHMOND, VIRGINIA 23288
804- 285-3210 FAX 804-285-3367
E-Mail: usaha@usaha.org
Web Site: www.usaha.org
 
May 22, 2007
Honorable Michael Johanns
Secretary of Agriculture
U.S. Department of Agriculture
Room 200 Jamie Whitten Federal Building
Washington, D.C. 20250
 
Dear Mr. Secretary:
 
The United States Animal Health Association (USAHA), wishes to express its encouragement to you and the Department of Agriculture to appeal the litigation surrounding private testing for Bovine Spongiform Encephalopathy. We hope you will strongly consider this as you work with the Office of General Counsel on this suit.
 
To support this appeal, we offer that this sets a detrimental precedence on USDA's ability to regulate disease and testing processes in animal agriculture. As we appreciate the entrepreneurial spirit of Creekstone, the larger scale implications could lead to devastating impacts for food animal production in this country as itrelates to animal health. We do feel that private testing could hamper animal health officials' ability to locate disease occurrences, and exercise proper practices to trace, control and eliminate them. As you are aware, there are a number of factors that raise concern among animal health leaders and diagnosticians. We encourage you to thoroughly consider those upon your decision to appeal. We do recognize this is now a matter of the courts, and trust that our ability to safeguard animal health is not compromised as a result of this litigation. Please let us know if there is any further support we can provide.
 
Sincerely,
 
Lee M. Myers
President, U.S. Animal Health Association
Cc: Dr. John Clifford
 
===============================
 
 
USA MAD COW STRAIN MORE VIRULENT TO HUMANS THAN UK STRAIN
 
18 January 2007 - Draft minutes of the SEAC 95 meeting (426 KB) held on 7 December 2006 are now available.
 
snip...
 
64. A member noted that at the recent Neuroprion meeting, a study was presented showing that in transgenic mice BSE passaged in sheep may be more virulent and infectious to a wider range of species than bovine derived BSE.
 
Other work presented suggested that BSE and bovine amyloidotic spongiform encephalopathy (BASE) MAY BE RELATED. A mutation had been identified in the prion protein gene in an AMERICAN BASE CASE THAT WAS SIMILAR IN NATURE TO A MUTATION FOUND IN CASES OF SPORADIC CJD.
 
snip...
 
http://www.seac.gov.uk/minutes/95.pdf
 
3:30 Transmission of the Italian Atypical BSE (BASE) in Humanized Mouse
 
Models Qingzhong Kong, Ph.D., Assistant Professor, Pathology, Case Western Reserve University
 
Bovine Amyloid Spongiform Encephalopathy (BASE) is an atypical BSE strain discovered recently in Italy, and similar or different atypical BSE cases were also reported in other countries. The infectivity and phenotypes of these atypical BSE strains in humans are unknown. In collaboration with Pierluigi Gambetti, as well as Maria Caramelli and her co-workers, we have inoculated transgenic mice expressing human prion protein with brain homogenates from BASE or BSE infected cattle. Our data shows that about half of the BASE-inoculated mice became infected with an average incubation time of about 19 months; in contrast, none of the BSE-inoculated mice appear to be infected after more than 2 years.
 
***These results indicate that BASE is transmissible to humans and suggest that BASE is more virulent than classical BSE in humans.***
 
6:30 Close of Day One
 
 
http://www.healthtech.com/2007/tse/day1.asp
 
Diagnosis and Reporting of Creutzfeldt-Jakob Disease
Singeltary, Sr et al. JAMA.2001; 285: 733-734.
 
http://jama.ama-assn.org/http://www.neurology.org/cgi/eletters/60/2/176#535
 
BRITISH MEDICAL JOURNAL
BMJ
vCJD in the USA * BSE in U.S.
15 November 1999
 
http://www.bmj.com/cgi/eletters/319/7220/1312/b#5406
 
BMJ
 
U.S. Scientist should be concerned with a CJD epidemic in the U.S., as
well...
 
2 January 2000
 
http://www.bmj.com/cgi/eletters/320/7226/8/b#6117
 
JOURNAL OF NEUROLOGY
MARCH 26, 2003
 
RE-Monitoring the occurrence of emerging forms of Creutzfeldt-Jakob disease in the United States
 
Email Terry S. Singeltary:
flounder@wt.net
 
I lost my mother to hvCJD (Heidenhain Variant CJD). I would like to comment on the CDC's attempts to monitor the occurrence of emerging forms of CJD. Asante, Collinge et al [1] have reported that BSE transmission to the 129-methionine genotype can lead to an alternate phenotype that is indistinguishable from type 2 PrPSc, the commonest sporadic CJD. However, CJD and all human TSEs are not reportable nationally. CJD and all human TSEs must be made reportable in every state and internationally. I hope that the CDC does not continue to expect us to still believe that the 85%+ of all CJD cases which ar sporadic are all spontaneous, without route/source. We have many TSEs i the USA in both animal and man. CWD in deer/elk is spreading rapidly and CWD does transmit to mink, ferret, cattle, and squirrel monkey by intracerebral inoculation. With the known incubation periods in other TSEs, oral transmission studies of CWD may take much longer. Every victim/family of CJD/TSEs should be asked about route and source of this agent. To prolong this will only spread the agent and needlessly exposeothers. In light of the findings of Asante and Collinge et al, there should be drastic measures to safeguard the medical and surgical arena from sporadic CJDs and all human TSEs. I only ponder how many sporadic CJDs in the USA are type 2 PrPSc?
 
http://www.neurology.org/cgi/eletters/60/2/176#535
 
doi:10.1016/S1473-3099(03)00715-1
Copyright © 2003 Published by Elsevier Ltd.
Newsdesk
 
Tracking spongiform encephalopathies in North America
Xavier Bosch
Available online 29 July 2003.
Volume 3, Issue 8, August 2003, Page 463
 
 
"My name is Terry S Singeltary Sr, and I live in Bacliff, Texas. I lost my mom to hvCJD (Heidenhain variant CJD) and have been searching for answers ever since. What I have found is that we have not been told the truth. CWD in deer and elk is a small portion of a much bigger problem."
 
............................
 
 
http://www.thelancet.com/journals/laninf/article/PIIS1473309903007151/fulltext
 
http://download.thelancet.com/pdfs/journals/1473-3099/PIIS1473309903007151.pdf
 
SEE STEADY INCREASE IN SPORADIC CJD IN THE USA FROM 1997 TO 2006. SPORADIC CJD CASES TRIPLED, with phenotype of 'UNKNOWN' strain growing. ...
 
http://www.cjdsurveillance.com/resources-casereport.html
 
There is a growing number of human CJD cases, and they were presented last
week in San Francisco by Luigi Gambatti(?) from his CJD surveillance
collection.
 
He estimates that it may be up to 14 or 15 persons which display selectively
SPRPSC and practically no detected RPRPSC proteins.
 
http://www.fda.gov/ohrms/dockets/ac/06/transcripts/1006-4240t1.htm
 
http://www.fda.gov/ohrms/dockets/ac/06/transcripts/2006-4240t1.pdf
 
[Docket No. FSIS-2006-0011] FSIS Harvard Risk Assessment of Bovine
Spongiform Encephalopathy (BSE)
 
http://www.fsis.usda.gov/OPPDE/Comments/2006-0011/2006-0011-1.pdf
 
[Docket No. 03-025IFA] FSIS Prohibition of the Use of Specified Risk Materials for Human Food and Requirement for the Disposition of Non-Ambulatory Disabled Cattle
 
03-025IFA
03-025IFA-2
 
http://www.fsis.usda.gov/OPPDE/Comments/03-025IFA/03-025IFA-2.pdf
 
THE SEVEN SCIENTIST REPORT ***
 
http://www.fda.gov/ohrms/dockets/dockets/02n0273/02n-0273-EC244-Attach-1.pdf
 
 
Sporadic creutzfeldt-jakob disease in two adolescents (sCJD, the big lie)
Date: May 28, 2007 at 7:58 am PST
 
http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0705&L=sanet-mg&T=0&P=25276
 
IN A NUT SHELL ;
 
(Adopted by the International Committee of the OIE on 23 May 2006)
 
11. Information published by the OIE is derived from appropriate declarations made by the official Veterinary Services of Member Countries. The OIE is not responsible for inaccurate publication of country disease status based on inaccurate information or changes in epidemiological status or other significant events that were not promptly reported to then Central Bureau............
 
http://www.oie.int/eng/Session2007/RF2006.pdf
 
 
Audit Report
Animal and Plant Health Inspection Service
Bovine Spongiform Encephalopathy (BSE) Surveillance Program ­ Phase II
and
Food Safety and Inspection Service
 
Controls Over BSE Sampling, Specified Risk Materials, and Advanced Meat
Recovery Products - Phase III
 
Report No. 50601-10-KC January 2006
 
Finding 2 Inherent Challenges in Identifying and Testing High-Risk Cattle
Still Remain
 
http://www.usda.gov/oig/webdocs/50601-10-KC.pdf
 
 
Report to Congressional Requesters:
February 2005:
Mad Cow Disease:
 
FDA's Management of the Feed Ban Has Improved, but Oversight Weaknesses
Continue to Limit Program Effectiveness:
 
[Hyperlink, http://www.gao.gov/cgi-bin/getrpt?GAO-05-101]:
 
http://www.gao.gov/htext/d05101.html
 
http://www.gao.gov/highlights/d05101high.pdf
 
January 2002 MAD COW DISEASE Improvements in the Animal Feed Ban and
Other Regulatory Areas Would Strengthen U.S. Prevention Efforts GAO-02-183
 
http://www.gao.gov/new.items/d02183.pdf
 
OIE BSE RECOMMENDATION FOR USA, bought and paid for by your local cattle
dealers i.e. USDA
 
Date: May 14, 2007 at 9:00 am PST
 
http://ranchers.net/forum/viewtopic.php?t=18748
 
What Do We Feed to Food-Production Animals? A Review of Animal Feed
Ingredients and Their Potential Impacts on Human Health
 
Date: May 24, 2007 at 6:59 am PST
 
http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0705&L=sanet-mg&T=0&P=22301
 
The Economic Impact of B.S.E. on the U.S. Beef Industry: BY NOT TESTING TO
FIND
 
Date: May 6, 2007 at 3:05 pm PST
 
http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0705&L=sanet-mg&T=0&P=4687
 
SCRAPIE UPDATE USA AS OF MARCH 2007 NOR98 INCLUDED
 
Date: May 9, 2007 at 6:43 pm PST
 
http://www.aphis.usda.gov/animal_health/animal_diseases/
scrapie/downloads/monthly_scrapie_rpt.pps
 
http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0705&L=sanet-mg&T=0&P=6721
 
http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0704&L=sanet-mg&T=0&P=8315
 
 
LIKE LAMBS TO SLAUGHTER
 
http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0704&L=sanet-mg&T=0&P=11598
 
Scrapie Agent (Strain 263K) Can Transmit Disease via the ORAL Route after
Persistence in Soil over Years
 
Date: May 16, 2007 at 10:01 am PST
 
http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0705&L=sanet-mg&T=0&P=15481
 
Colorado Surveillance Program for Chronic Wasting Disease Transmission to
Humans (TWO SUSPECT CASES)
 
Date: Wed, 4 Apr 2007 16:22:22 -0500
 
http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0704&L=sanet-mg&T=0&P=1165
 
Subject: Re: FOIA REQUEST FOR ATYPICAL TSE INFORMATION ON VERMONT SHEEP
 
Sent: Monday, April 02, 2007 2:37 PM
 
http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0704&L=sanet-mg&T=0&P=816
 
EXPORTATION AND IMPORTATION OF ANIMALS AND ANIMAL PRODUCTS:
BSE; MRR AND IMPORTATION OF COMMODITIES, 65758-65759 [E6-19042]
 
http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0701&L=sanet-mg&T=0&P=3854
 
http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0611&L=sanet-mg&T=0&P=3381
 
http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0703&L=sanet-mg&T=0&P=498
 
http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0702&L=sanet-mg&T=0&P=10277
 
http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0701&L=sanet-mg&T=0&P=9972
 
http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0703&L=sanet-mg&T=0&P=4492
 
http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0703&L=sanet-mg&T=0&P=2583
 
http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0703&L=sanet-mg&T=0&P=2470
 
HUMAN and ANIMAL TSE Classifications i.e. mad cow disease and the UKBSEnvCJD
only theory
 
 
TSEs have been rampant in the USA for decades in many species, and they all have been rendered and fed back to animals for human/animal consumption. I propose that the current diagnostic criteria for human TSEs only enhances and helps the spreading of human TSE from the continued belief of the UKBSEnvCJD only theory in 2005. With all the science to date refuting it, to continue to validate this myth, will only spread this TSE agent through a multitude of potential routes and sources i.e. consumption, surgical, blood, medical, cosmetics etc. I propose as with Aguzzi, Asante, Collinge, Caughey, Deslys, Dormont, Gibbs, Ironside, Manuelidis, Marsh, et al and many more, that the world of TSE Tranmissible Spongiform Encephalopathy is far from an exact science, but there is enough proven science to date that this myth should be put to rest once and for all, and that we move forward with a new classification for human and animal TSE that would properly identify the infected species, the source species, and then the route. This would further have to be broken down to strain of species and then the route of transmission would further have to be broken down.
 
Accumulation and Transmission are key to the threshold from subclinical to clinical disease, and of that, I even believe that physical and or blunt trauma may play a role of onset of clinical symptoms in some cases, but key to all this, is to stop the amplification and transmission of this agent, the spreading of, no matter what strain. BUT, to continue with this myth that the U.K. strain of BSE one strain in cows, and the nv/v CJD, one strain in humans, and that all the rest of human TSE is one single strain i.e. sporadic CJD (when to date there are 6 different phenotypes of sCJD), and that no other animal TSE transmits to humans, to continue with this masquerade will only continue to spread, expose, and kill, who knows how many more in the years and decades to come. ONE was enough for me, My Mom, hvCJD, DOD 12/14/97 confirmed, which is nothing more than another mans name added to CJD, like CJD itself, Jakob and Creutzfeldt, or Gerstmann-Straussler-Scheinker syndrome, just another CJD or human TSE, named after another human. WE are only kidding ourselves with the current diagnostic criteria for human and animal TSE, especially differentiating between the nvCJD vs the sporadic CJD strains and then the GSS strains and also the FFI fatal familial insomnia strains or the ones that mimics one or the other of those TSE? Tissue infectivity and strain typing of the many variants of the human and animal TSEs are paramount in all variants of all TSE. There must be a proper classification that will differentiate between all these human TSE in order to do this. With the CDI and other more sensitive testing coming about, I only hope that my proposal will some day be taken seriously.
 
My name is Terry S. Singeltary Sr. and I am no scientist, no doctor and have no PhDs, but have been independently researching human and animal TSEs since the death of my Mother to the Heidenhain Variant of Creutzfeldt Jakob Disease on December 14, 1997 'confirmed'. ...TSS
 
 
UNITED STATES DISTRICT COURT
FOR THE DISTRICT OF COLUMBIA
CREEKSTONE FARMS PREMIUM BEEF,
L.L.C.,
Plaintiff,
v.
U.S. DEPARTMENT OF AGRICULTURE,
et al.,
Defendants.
:::::::::::
Civil Action No. 06-0544 (JR)
 
snip...
 
JAMES ROBERTSON
United States District Judge
 
The government's additional argument, that private testing 14 somehow would interfere with USDA's surveillance program, is unexplained and therefore rejected. Of greater concern is the possibility that private testing 15 could produce a false positive result, which might trigger unnecessary public alarm. USDA has asserted this possibility as a reason to avoid private testing. Indeed, the Bio-Rad kits that Creekstone proposes using are used throughout the world, including as part of the USDA's own surveillance testing. - 18 -
 
https://ecf.dcd.uscourts.gov/cgi-bin/show_public_doc?2006cv0544-22
 
Terry S. Singeltary Sr.
P.O. Box 42
Bacliff, Texas USA 77518

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